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Fig. 6 | Cellular & Molecular Biology Letters

Fig. 6

From: rt269L-Type hepatitis B virus (HBV) in genotype C infection leads to improved mitochondrial dynamics via the PERK–eIF2α–ATF4 axis in an HBx protein-dependent manner

Fig. 6

rt269L leads to increased HBx protein stability. A Immunofluorescence images of HBx-GFP expression in HepG2 cells cotransfected with mock, rt269L, and rt269I whole genome plasmids. B Immunohistochemical (IHC) analysis of HBx expression in paraffin-embedded liver tissues (magnification 100 ×, n = 5 per group). C Western blot analysis of HBx-GFP in HepG2 cells cotransfected with mock, rt269L, or rt269I vector. D Immunofluorescence images of HBx-GFP expression in HepG2 cells cotransfected with mock, rt269L (genotype A or C), and rt269I (genotype A or C) whole genome plasmids. E Western blot analysis of HBx-GFP in HepG2 cells cotransfected with mock, rt269L (genotype A or C), or rt269I (genotype A or C) vector. F Luciferase reporter assay with the pHBV-X/EnhI-Luc construct composed of the X promoter region and enhancer I at 48 h. The relative intensity of HBx/GAPDH staining was analyzed. **p < 0.01, ***p < 0.001

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Cellular & Molecular Biology Letters

ISSN: 1689-1392

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